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INJURY REPAIR

Tendon & Soft Tissue Healing Protocol

3-Phase Recovery

WEEKS 1-2
Activation
Restart healing, reduce inflammation
WEEKS 3-6
Remodeling
Build collagen, restore tissue
WEEKS 7-10+
Integration
Consolidate, return to function
The Protocol
The Core Compounds
The Systemic Healer

BPC-157

Restores angiogenesis and perfusion. Activates VEGFR2-Akt-eNOS signaling, accelerates fibroblast migration and upregulates Type I/III collagen.

You'll Notice
  • Pain reduction 30-50% within 1-2 weeks
  • Better range of motion and tissue glide
  • Accelerated wound healing
Cell Migration Orchestrator

TB-500

Regulates actin dynamics for efficient cell migration and proper spatial organization. Reduces adhesions between tissue planes.

You'll Notice
  • Morning stiffness and first-step pain decrease
  • Cleaner tendon glide, less catching
  • Improved range of motion
Collagen Architecture Optimization

GHK-Cu

Regulates matrix metalloproteinases to break down disorganized collagen while upregulating organized synthesis. Orchestrates transition from reactive scar tissue to functional matrix.

You'll Notice
  • Tissue elasticity and functional quality improve
  • Scar color and texture normalize over 6-12 weeks
  • Thick, ropey tissue softens and becomes pliable
Anti-Inflammatory Master Switch

KPV

Directly blocks NF-κB—the master transcription factor orchestrating inflammatory gene expression—with precision and without systemic immunosuppression.

You'll Notice
  • Hot, swollen tissues calm within 3-7 days
  • Tendons become load-tolerant
  • Healing progresses without inflammation
The Power Supply

NAD+

Powers cellular energy systems driving all tissue regeneration. Fuels metabolic processes behind collagen production, DNA repair, and cellular turnover—giving cells the energy reserves needed to execute healing.

You'll Notice
  • Enhanced cellular vitality and repair response
  • Accelerated recovery across all systems
  • Improved stress resistance to daily damage
Repairing Broken Signals
Stalled Healing

Angiogenesis stops, fibroblasts go dormant— tissue lack nutrients and healing signals

BPC-157

Restarts perfusion and cellular activation through VEGFR2-Akt-eNOS signaling

Limited Cell Migration

Cells can't move to injury site, tissue planes stick together, mobility restricted

TB-500

Enables cell migration and tissue organization by regulating actin dynamics

Chronic Inflammation

Overactive immune response creates heat, swelling, pain—blocks tissue from rebuilding

KPV

Calms inflammation without suppressing repair by directly blocking NF-κB

Poor Tissue Quality

Disorganized collagen forms scar tissue—weak, inflexible, prone to re-injury

GHK-CU

Optimizes collagen architecture by regulating matrix metalloproteinases

Contraindications
  • Active malignancy or proliferative retinopathy → avoid BPC-157 & TB-500 (angiogenic)
  • Wilson's disease → avoid GHK-Cu
  • Pregnancy / breastfeeding → insufficient safety data
References